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Tehran: Chemical Warfare Research Center (2000)

Stage 1: Depleted Stores (Often Asymptomatic) Liver B12 stores decline

To confirm that the observed effects were mediated through direct GLP-1R activation, or indeed, that they are not secondary autocrine effects of insulin, we performed 2-NBDG uptake experiments in the presence of the GLP1R antagonist Exendin (939) and the potassium channel opener, diazoxide

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