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Rodent studies have revealed that hexarelin can increase cardiac output, stroke volume, and oxygen and blood flow in mice after myocardial infarction, while protecting rat cardiomyocytes via the interleukin-1 signaling pathway following myocardial ischemia/reperfusion (I/R) injury [3, 4]

When we treated the slices with 100 nM tertiapin-Q, a GIRK channel blocker, membrane potential was depolarized by 2.90.3 mV (from 52.42.1 to 49.51.9 mV, n =4) in four of seven POMC neurons (50%) (Fig