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As previously described the viral infection induced an increase in ROS production directly in CNS cells or indirectly by the infiltrated activated immune system cells, which use ROS release as mechanism to control the infection (Figure 3)

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Huang J, Yi H, Zhao C, Zhang Y, Zhu L, Liu B, He P, Zhou M

However, the residual risk in these patients remains high, which emphasizes a need for novel therapies or novel strategies to facilitate a reduction in residual risk

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