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Although still uncorroborated by well-defined translational substantiation, the GLP1-RAs-induced upregulation of ACE2 could ameliorate lung injury, antagonizing the reduction of ACE2 expression levels that are hallmarks of SARS-CoV-2 infection progression [65] and precluding the over-activated immune response critical for acute respiratory distress syndrome [66] (Figure 1)

Cuttica CM, Briata IM, DeCensi A

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