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The ground-breaking finding that mice expressing dominant-negative N-cadherin, an adherent junction protein, develop spontaneous (not induced using chemicals or infections known to induce colitis in mice) inflammation in the gut epithelium further supports the key role of cell-to-cell adhesion, especially in its capacity to contribute to overall mucosal barrier function in regulating mucosal immune homeostasis

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Targeting the GLP-1/GLP-1R axis to treat osteoarthritis: A new opportunity

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Chemical properties of lipids strongly affect the kinetics of the membrane-induced aggregation of -synuclein