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it was a two-pronged metabolic strike team, stimulating both GLP-1 and GIP receptors

Friedrich et al., 2018

Several plausible explanations include: insufficient autophagy suppression in patient tumors by HCQ, the dual role of autophagy inhibition in suppressing tumor metabolism and proliferation while potentially promoting metastasis, and the current lack of suitable pharmacodynamic biomarkers (66, 106, 107)

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Additionally, progression through the dose-escalation protocol affects resultspatients who cannot tolerate higher maintenance doses (10 mg or 15 mg) due to adverse effects may experience less weight loss than those reaching maximum doses