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This agent differs from earlier therapies that were designed to reduce myocardial loading or induce neurohumoral blockade

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The ferroptosis inhibitor liproxstatin-1 has been shown to reverse iron accumulation, endothelial damage, and increased atherosclerosis in Jak2VF chimeric mice and VFEpoR mice, 539 indicating a potential therapeutic target for mitigating erythrocytic ferroptosis-mediated cardiovascular risk