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Given the role of LoF mutations in ASD etiology, many of the therapeutics in development are designed to rescue the haploinsufficient effect of genes at the transcriptional, translational, and protein levels

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97 Proteins such as integrins and focal adhesion kinase (FAK) are involved in these pathways, linking the extracellular matrix to the intracellular cytoskeleton and transmitting mechanical signals that promote muscle growth and adaptation