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This collection of symptoms is collectively known as Burnside-Butler syndrome ( The PWS critical region is located between BP2-BP3 and contains genes that are crucially involved in growth processes, neurodevelopmental and hormonal regulation
In other contexts, genetic variation is known to have an important role in treatment response, both for intended and adverse events 6,7,8
For example, in short term infusion studies in humans, GIP increases the level of osteopontin, which is considered to be a pro-atherogenic cytokine [26]
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