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In line with the effects of selenium metabolism and selenoproteins in chondrogenic progenitor cells observed in vitro, deficient uptake of selenium severely affected chondrogenic differentiation of mesenchymal lineage cells and thus endochondral ossification in mice 64
Dysregulated mTORC1 signaling impairs cell growth, synaptic plasticity, autophagy and pruning, resulting in excessive dendritic spines and hyperactive circuits (Shang et al
In athletic performance, mitochondrial efficiency directly influences endurance, recovery speed, and overall work capacity
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