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It has been hypothesized that induction of chronic oxidative stress mediates the late effects of ionizing radiation

Results vary, but gradual improvements are seen with consistent use

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However, recent work has shown that, in addition to using histones as substrates for deacetylation, HDACs can also inhibit the acetylation of non-histone proteins and modulate diverse neurobiological processes [59, 60]

Anti-inflammatory action of statins in cardiovascular disease: the role of inflammasome and toll-like receptor pathways