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Figure 7D) also suggests that accumulation in ER subdomains is a feature of G6PD5.4 and largely independent of the presence of PGD2 or PGL2

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In pancreatic ductal carcinoma cells, suppression of GPx1 expression promoted ROS-induced Akt signaling (measured by Akt phosphorylation) to drive an epithelial-mesenchymal transition that is associated with enhanced tumor growth, whereas overexpression of GPx1 decreased the phenotypic changes in pancreatic ductal carcinoma cells by decreasing ROS-mediated pro-survival signaling in these cells [134]

Anti-atherosclerotic effects of the glucagon-like peptide-1 (GLP-1) based therapies in patients with type 2 diabetes mellitus: A meta-analysis

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