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Ligand engagement promotes receptor autophosphorylation on intracellular tyrosine residues and recruitment of adaptor proteins such as IRS and Shc

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Meanwhile, in January 1996 the first rodent study suggested that GLP-1 could be a central regulator of feeding behavior and satiety based on intracerebroventricular injection studies [4], but soon thereafter the results of other rodent studies using peripherally injected GLP-1 argued against an appetite effect [5]

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