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SLC15A3 plays a crucial role in pulmonary fibrosis by regulating macrophage oxidative stress

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The McCoy 2013 paper extended this work by introducing N- and C-terminal modifications designed to enhance metabolic stability and hydrophobicity, ultimately producing Dihexa (N-hexanoic-Tyr-Ile-(6) aminohexanoic amide) as an orally active, BBB-permeable analog [2]

The formation of multiple noncovalent interactions was then investigated by FT-IR spectroscopy as presented in Supplementary Fig