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Traditional treatments like prescription drugs may mask the pain temporarily, but they dont address the root cause of nerve damage

Their ability to simultaneously address metabolic risk factors, inflammation, and cellular proliferation positions them as one of the most promising drug classes for interrupting SLD progression (Figure 2)

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The central hypothesis is that EECs contribute to the pathogenesis of inflammatory bowel diseases (IBD) due to their role in endocrine-immune crosstalk, particularly via GLP-1's influence on intraepithelial lymphocytes and metabolic-inflammatory responses

Options include staying at your current dose longer before increasing, temporarily reducing to your previous dose level, implementing more aggressive side effect management strategies, trying anti-nausea medications, or in some cases discontinuing treatment