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DOI: 10.1016/j.freeradbiomed.2010.12.029 50
VEGF-stimulating compounds can, in principle, support the vascularization of existing tumors or accelerate the growth of pre-malignant lesions
Pathophysiological basis]
The interaction hypothesis rests on the individual mechanistic profiles of the components: BPC-157 and TB-500 address pro-angiogenic and actin-based tissue reconstruction
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