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The fact that inhibition of either proteasome or lysosome can partially but not completely block the degradation of ACE2 suggests that both organelles are critical to ACE2 catabolism, and the role of other forms of post-translational modification in ACE2 catabolism warrants further investigation, writes the team

2016 Behavioral Risk Factor Surveillance System Results. Available at: 19 Pierce, John P, et al

Salvi, S.S

3.2 Mitophagy mediated inflammation in fibrotic diseases Chronic inflammation is a key feature of fibrotic diseases, characterized by the activation of inflammasomes such as NLR family pyrin domain containing 3 (NLRP3), inflammatory signaling pathways like nuclear factor-kappa B (NF-B), and the upregulation of pro-inflammatory cytokines, along with the recruitment and polarization of immune cells